This results in thickened airway walls and increased smooth muscle. The airway wall of patients with asthma is characterized by increased smooth muscle mass, mucous gland hypertrophy and vascular congestion. Fibrosis or Scarring: ongoing inflammation in the airways can lead to development of scar tissue and “tissue remodelling”.Mucus: mucus production is normally a protective response, but in severe asthma, it is excessive and can block the inside of the airways – preventer medicines also reduce mucus.Preventer or controller medicines work by reducing the inflammation that causes the swelling. Inflammation / Swelling: the inside walls of the airways are often swollen and inflamed, leaving less space inside.They do not treat other aspects of asthma described below. Reliever inhalers relieve symptoms by relaxing these muscles in the airways for a few hours. In people with asthma, this muscle is often “twitchy” and contracts more easily and more strongly than in people who do not have asthma. Airway Muscle: the thin layer of muscle within the wall of an airway can contract to make it tighter and narrower.1 It is characterized by intermittent airflow obstruction and airway inflammation, producing symptoms of chest tightness, wheezing, and cough. Brochures or posters from Asthma Australia or National Asthma CouncilĮxplain to the patient the main changes to the airways that occur in asthma, and that all these can happen together: asthma is a chronic disease of the airways affecting over 24 million people in the United States ().These resources are relevant to asthma generally: Systematic & Multidimensional Assessment ResourcesĬommence patient education with an overview or review of respiratory anatomy and asthma physiology using visual aids. ![]() Alternative Diagnosis & Co-Morbidities in Paediatrics.Staffing & Multidisciplinary Team Approach.Cardiovascular Disease & Metabolic Disease. ![]() ![]()
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